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The Combined Functions of Proapoptotic Bcl-2 Family Members Bak and Bax Are Essential for Normal Development of Multiple Tissues

Molecular Cell · 2000 · Vol. 6(6) · pp. 1389–1399

Tullia Lindsten✉(Cancer Research Institute)Andrea J. Ross(Emory University)Ayala King(Cancer Research Institute)Wei‐Xing Zong(Cancer Research Institute)Jeffrey C. Rathmell(Cancer Research Institute)Helena Shiels(University of Chicago)Eugen Ulrich(Cancer Research Institute)Katrina G. Waymire(Emory University)Patryce L. Mahar(Emory University)Kenneth A. Frauwirth(Cancer Research Institute)Yifeng Chen(University of Pennsylvania)Michael C. WeiVicki M. Eng(University of Pennsylvania)David M. Adelman(University of Pennsylvania)M. Celeste Simon(Howard Hughes Medical Institute)Averil Ma(University of Chicago)Jeffrey A. Golden(University of Pennsylvania)Gérard I. Evan(Cancer Research Institute)Stanley J. KorsmeyerGrant R. MacGregor(Emory University)Craig B. Thompson(University of Pennsylvania)

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Cell death mechanisms and regulationBiologyBcl-2 familyApoptosisCell biologyCancer researchGeneticsProgrammed cell death

MeSH terms

Abnormalities, MultipleAnimalsBrainCells, CulturedCrosses, GeneticEtoposideFemaleEmbryonic and Fetal DevelopmentHematopoietic Stem CellsHistocytochemistryKidneyLymphoid TissueMaleMembrane ProteinsPhenotype

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Control of mitochondrial apoptosis by the Bcl-2 family