articleTop 1% cited
Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell death
Cell · 1993 · Vol. 74(4) · pp. 609–619
Zoltán N. Oltval✉(Howard Hughes Medical Institute)Curt Milliman(Howard Hughes Medical Institute)Stanley J. Korsmeyer(Howard Hughes Medical Institute)
Cell death mechanisms and regulationImmune Response and InflammationNF-κB Signaling PathwaysBiologyProgrammed cell deathBcl-2-associated X proteinApoptosisExonRNA splicingMolecular biologyRepressorCell biologyAlternative splicing
MeSH terms
Amino Acid SequenceAnimalsBase SequenceCell LineCloning, MolecularHumansMolecular Sequence DataProto-Oncogene ProteinsBlotting, WesternRestriction MappingPolymerase Chain ReactionConserved SequenceApoptosisSequence Homology, Amino AcidProto-Oncogene Proteins c-bcl-2
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