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Mitochondrial Dysfunction Induces Senescence with a Distinct Secretory Phenotype

Cell Metabolism · 2015 · Vol. 23(2) · pp. 303–314

Christopher D. Wiley(Buck Institute for Research on Aging)Michael C. Velarde(Buck Institute for Research on Aging)Pacôme Lecot(Buck Institute for Research on Aging)Su Liu(Buck Institute for Research on Aging)Ethan A. Sarnoski(Buck Institute for Research on Aging)Adam Freund(Buck Institute for Research on Aging)Kotaro Shirakawa(Gladstone Institutes)Hyung W. Lim(Gladstone Institutes)Sonnet S. Davis(Buck Institute for Research on Aging)Arvind Ramanathan(Buck Institute for Research on Aging)Akos A. Gerencser(Buck Institute for Research on Aging)Eric Verdin(Gladstone Institutes)Judith Campisi✉(Buck Institute for Research on Aging)

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Telomeres, Telomerase, and Senescence Mitochondrial Function and Pathology Autophagy in Disease and TherapySenescencePhenotypeCell biologyBiologyMitochondrionMitosisMitochondrial DNAAdipogenesisAMPKCell growth

MeSH terms

DNA Polymerase gammaAdenylate KinaseAnimalsDNA-Directed DNA PolymeraseEnzyme ActivationMitochondriaNADPhenotypeTumor Suppressor Protein p53Cellular SenescenceSirtuinsMice

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Telomere Biology and Human Phenotype