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CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer

Nature Immunology · 2009 · Vol. 11(2) · pp. 155–161

Cameron R. Stewart✉Lynda M. Stuart(Harvard University)Kim Wilkinson(Harvard University)Janine M. van Gils(Harvard University)Jiusheng Deng(Harvard University)Annett Halle(University of Massachusetts Chan Medical School)Katey J. Rayner(Harvard University)Laurent Boyer(Massachusetts General Hospital)Ruiqin Zhong(Washington University in St. Louis)William A. Frazier(Washington University in St. Louis)Adam Lacy‐Hulbert(Massachusetts General Hospital)Joseph El Khoury(Harvard University)Douglas T. Golenbock(University of Massachusetts Chan Medical School)Kathryn J. Moore(Harvard University)

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Immune Response and Inflammation Neuroinflammation and Neurodegeneration Mechanisms Cholesterol and Lipid MetabolismInflammationScavenger receptorTLR2CD36Toll-like receptorInnate immune systemReceptorCell biologyTLR4Pattern recognition receptor

MeSH terms

AnimalsCell LineHumansInflammationLipoproteins, LDLMice, Inbred C57BLBlotting, WesternSignal TransductionGene ExpressionAmyloid beta-PeptidesMicrogliaMice, KnockoutChemokinesCD36 AntigensReverse Transcriptase Polymerase Chain Reaction

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Physiology of Microglia