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Natural oligomers of the amyloid-β protein specifically disrupt cognitive function

Nature Neuroscience · 2004 · Vol. 8(1) · pp. 79–84
James ClearyDominic M. WalshJ. HofmeisterGanesh M. ShankarMichael A. KuskowskiDennis J. SelkoeKaren H. Ashe
Alzheimer's disease research and treatmentsComputational Drug Discovery MethodsProtein Structure and DynamicsNeuroscienceCognitionDementiaAmyloid (mycology)DiseaseAmyloidosisAlzheimer's diseaseFunction (biology)Amyloid betaChemistry

MeSH terms

AnimalsCognition DisordersConditioning, OperantCricetulusFemaleCricetinaeHumansInjections, IntraventricularMaleAmyloid beta-PeptidesCHO CellsCulture Media, ConditionedRats, Sprague-DawleyRats
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