articleTop 1% cited
Natural oligomers of the amyloid-β protein specifically disrupt cognitive function
Nature Neuroscience · 2004 · Vol. 8(1) · pp. 79–84
James Cleary✉Dominic M. Walsh(Brigham and Women's Hospital)J. Hofmeister(Geriatric Research Education and Clinical Center)Ganesh M. Shankar(Harvard University)Michael A. Kuskowski(University of Minnesota)Dennis J. Selkoe(Harvard University)Karen H. Ashe(University of Minnesota)
Alzheimer's disease research and treatmentsComputational Drug Discovery MethodsProtein Structure and DynamicsNeuroscienceCognitionDementiaAmyloid (mycology)DiseaseAmyloidosisAlzheimer's diseaseFunction (biology)Amyloid betaChemistry
MeSH terms
AnimalsCognition DisordersConditioning, OperantCricetulusFemaleCricetinaeHumansInjections, IntraventricularMaleAmyloid beta-PeptidesCHO CellsCulture Media, ConditionedRats, Sprague-DawleyRats
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References
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Trends in Neurosciences · 2001 · 994 citations
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Neurobiology of Aging · 1991 · 826 citations
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Annals of Neurology · 1988 · 1,224 citations
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Trends in Pharmacological Sciences · 1991 · 2,515 citations
Soluble pool of A? amyloid as a determinant of severity of neurodegeneration in Alzheimer's disease
Annals of Neurology · 1999 · 1,797 citations
Phenolic anti-inflammatory antioxidant reversal of Aβ-induced cognitive deficits and neuropathology
Neurobiology of Aging · 2001 · 510 citations
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