reviewTop 1% cited

Soluble oligomers of the amyloid β-protein impair synaptic plasticity and behavior

Behavioural Brain Research · 2008 · Vol. 192(1) · pp. 106–113

Alzheimer's disease research and treatments Neuroscience and Neuropharmacology Research Prion Diseases and Protein MisfoldingLong-term potentiationHippocampal formationNeuroscienceSynaptic plasticityDendritic spineChemistryAmyloid betaAlzheimer's diseaseAmyloid (mycology)Cell biology

MeSH terms

Alzheimer DiseaseAnimalsCerebral CortexHumansModels, NeurologicalSynaptic TransmissionNeuronal PlasticityPolymersSynapsesAmyloid beta-PeptidesLong-Term PotentiationDendritic Spines

Citations

1,006

FWCI

48.03

field-weighted impact

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100%

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References

Targeting small Aβ oligomers: the solution to an Alzheimer's disease conundrum?

Trends in Neurosciences · 2001 · 994 citations

Regulation of NMDA receptor trafficking by amyloid-β

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Tangles and plaques in nondemented aging and ?preclinical? Alzheimer's disease

Annals of Neurology · 1999 · 1,682 citations

Natural Oligomers of the Alzheimer Amyloid-β Protein Induce Reversible Synapse Loss by Modulating an NMDA-Type Glutamate Receptor-Dependent Signaling Pathway

Journal of Neuroscience · 2007 · 1,574 citations

Physical basis of cognitive alterations in alzheimer's disease: Synapse loss is the major correlate of cognitive impairment

Annals of Neurology · 1991 · 4,370 citations

Visualization of Aβ42(43) and Aβ40 in senile plaques with end-specific Aβ monoclonals: Evidence that an initially deposited species is Aβ42(43)

Neuron · 1994 · 1,756 citations

A quantitative morphometric analysis of the neuronal and synaptic content of the frontal and temporal cortex in patients with Alzheimer's disease

Journal of the Neurological Sciences · 1987 · 614 citations

The molecular pathology of Alzheimer's disease

Neuron · 1991 · 2,717 citations

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