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Stromal Elements Act to Restrain, Rather Than Support, Pancreatic Ductal Adenocarcinoma

Cancer Cell · 2014 · Vol. 25(6) · pp. 735–747

Andrew D. Rhim(Cancer Research Institute)Paul E. Oberstein(Columbia University Irving Medical Center)Dafydd Thomas(Columbia University Irving Medical Center)Emily T. Mirek(Cancer Research Institute)Carmine F. Palermo(Columbia University Irving Medical Center)Stephen A. Sastra(Columbia University Irving Medical Center)Erin N. Dekleva(Cancer Research Institute)Tyler Saunders(Johns Hopkins University)Carla Angelica Berrio Becerra(Columbia University Irving Medical Center)Ian Tattersall(Columbia University Irving Medical Center)C. Benedikt Westphalen(Columbia University Irving Medical Center)Jan Kitajewski(Columbia University Irving Medical Center)Maite G. Fernández‐Barrena(Mayo Clinic)Martín E. Fernández-Zapico(Mayo Clinic)Christine A. Iacobuzio–Donahue(Johns Hopkins University)Kenneth P. Olive✉(Columbia University Irving Medical Center)Ben Z. Stanger✉(Cancer Research Institute)

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Hedgehog Signaling Pathway Studies Pancreatic and Hepatic Oncology Research Tumors and Oncological CasesStromaStromal cellSmoothenedSonic hedgehogCancer researchHedgehogBiologyAdenocarcinomaAngiogenesisPancreatic cancer

MeSH terms

AnimalsAntibodies, MonoclonalDisease Models, AnimalHumansImmunoglobulin GMice, TransgenicPancreatic NeoplasmsRandom AllocationSignal TransductionStromal CellsDisease ProgressionCarcinoma, Pancreatic DuctalReceptors, Vascular Endothelial Growth FactorCell Line, TumorMice

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