article Open AccessTop 1% cited
Targeted disruption of the PU.1 gene results in multiple hematopoietic abnormalities.
The EMBO Journal · 1996 · Vol. 15(20) · pp. 5647–5658
Scott R. McKercher✉(Sanford Burnham Prebys Medical Discovery Institute)Bruce E. Torbett(Sanford Burnham Prebys Medical Discovery Institute)Kenton L. Anderson(Sanford Burnham Prebys Medical Discovery Institute)Gregory W. Henkel(Sanford Burnham Prebys Medical Discovery Institute)Deborah J. Vestal(Sanford Burnham Prebys Medical Discovery Institute)Hélène Baribault(Sanford Burnham Prebys Medical Discovery Institute)M Klemsz(Sanford Burnham Prebys Medical Discovery Institute)Ann J. Feeney(Sanford Burnham Prebys Medical Discovery Institute)G E Wu(Sanford Burnham Prebys Medical Discovery Institute)Christopher J. Paige(Sanford Burnham Prebys Medical Discovery Institute)R. Maki(Sanford Burnham Prebys Medical Discovery Institute)
Cell Adhesion Molecules ResearchImmune Response and InflammationImmunotherapy and Immune ResponsesBiologyHaematopoiesisMyeloidLineage (genetic)ImmunologyMolecular biologyGeneCell biologyStem cellGenetics
MeSH terms
Proto-Oncogene Protein Spi-1AnimalsB-LymphocytesBinding SitesCell DifferentiationDNAFlow CytometryHematopoiesisMacrophagesNeutrophilsProto-Oncogene ProteinsT-LymphocytesTrans-ActivatorsMice
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