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c-Myc and Cancer Metabolism

Clinical Cancer Research · 2012 · Vol. 18(20) · pp. 5546–5553
Donald M. MillerShelia D. ThomasAshraful IslamDavid E. MuenchKara C. Sedoris

Abstract

The processes of cellular growth regulation and cellular metabolism are closely interrelated. The c-Myc oncogene is a "master regulator" which controls many aspects of both of these processes. The metabolic changes which occur in transformed cells, many of which are driven by c-Myc overexpression, are necessary to support the increased need for nucleic acids, proteins, and lipids necessary for rapid cellular proliferation. At the same time, c-Myc overexpression results in coordinated changes in level of expression of gene families which result in increased cellular proliferation. This interesting duality of c-Myc effects places it in the mainstream of transformational changes and gives it a very important role in regulating the "transformed phenotype." The effects induced by c-Myc can occur either as a "primary oncogene" which is activated by amplification or translocation or as a downstream effect of other activated oncogenes. In either case, it appears that c-Myc plays a central role in sustaining the changes which occur with transformation. Although efforts to use c-Myc as a therapeutic target have been quite frustrating, it appears that this may change in the next few years.

Cancer, Hypoxia, and MetabolismBiochemical and Molecular ResearchCancer-related Molecular PathwaysProto-Oncogene Proteins c-mycOncogeneBiologyRegulatorCell biologyCell growthPhenotypeCancer cellRegulation of gene expressionGene

MeSH terms

Cell Transformation, NeoplasticGlutamineGlycolysisHumansMitochondriaNeoplasmsGene Expression Regulation, NeoplasticProto-Oncogene Proteins c-mycCell ProliferationMolecular Targeted Therapy
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