articleTop 1% cited
Molecular and chromosomal mechanisms of resistance to imatinib (STI571) therapy
Leukemia · 2002 · Vol. 16(11) · pp. 2190–2196
Andreas Hochhaus✉(Heidelberg University)Sebastian Kreil(University Hospital Heidelberg)A. Corbin(Oregon Health & Science University)Paul La Rosée(Oregon Health & Science University)MC Müller(University Hospital Heidelberg)Tanja Lahaye(Heidelberg University)Benjamin Hanfstein(Heidelberg University)Claudia SchochNicholas C.P. Cross(University of Southampton)Ute Berger(Heidelberg University)Harald Gschaidmeier(Novartis (Germany))Brian Druker(Oregon Health & Science University)Rüdiger Hehlmann(Heidelberg University)
Chronic Myeloid Leukemia TreatmentsEosinophilic Disorders and SyndromesChronic Lymphocytic Leukemia ResearchImatinibTyrosine kinasePhiladelphia chromosomeCancer researchABLChronic myelogenous leukemiabreakpoint cluster regionImatinib mesylateTyrosine-kinase inhibitorMyeloid leukemia
MeSH terms
Imatinib MesylateAntineoplastic AgentsBenzamidesChromosome AberrationsDNA Mutational AnalysisDNA, NeoplasmEnzyme InhibitorsHumansMutationNeoplasm Recurrence, LocalPiperazinesProtein-Tyrosine KinasesPyrimidinesLeukemia, Myelogenous, Chronic, BCR-ABL PositiveFusion Proteins, bcr-abl
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References
The P-loop — a common motif in ATP- and GTP-binding proteins
Trends in Biochemical Sciences · 1990 · 2,074 citations
The molecular biology of chronic myeloid leukemia
Blood · 2000 · 1,610 citations
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