review Open AccessTop 1% cited
Experimental Biology of Cerebral Hypoxia-Ischemia: Relation to Perinatal Brain Damage
Pediatric Research · 1990 · Vol. 27(4) · pp. 317–326
Robert C. Vannucci✉(Penn State Milton S. Hershey Medical Center)
Neonatal and fetal brain pathologyMetabolism and Genetic DisordersMitochondrial Function and PathologyNeurochemicalHypoxia (environmental)IschemiaBrain damageCerebral palsyMedicineNeuroscienceCerebral hypoxiaBrain ischemiaFetus
MeSH terms
AnimalsAnimals, NewbornBrain InjuriesCalciumHypoxia, BrainBrain IschemiaEnergy MetabolismFree RadicalsHomeostasisHumansInfant, NewbornSecond Messenger SystemsSignal TransductionNeurotransmitter Agents
Funding
- American Diabetes Association
- American Heart Association
- National Institutes of Health
Citations
431
FWCI
23.38
field-weighted impact
References
68
Percentile
100%
vs. same field & year
Citations per year
Cited by
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References
Radioimmunoassay for Cyclic Nucleotides
Journal of Biological Chemistry · 1972 · 2,032 citations
Effect of Ischemia on Known Substrates and Cofactors of the Glycolytic Pathway in Brain
Journal of Biological Chemistry · 1964 · 2,230 citations
Calcium Fluxes, Calcium Antagonists, and Calcium-Related Pathology in Brain Ischemia, Hypoglycemia, and Spreading Depression: A Unifying Hypothesis
Journal of Cerebral Blood Flow & Metabolism · 1989 · 1,239 citations
Delayed neuronal death in the gerbil hippocampus following ischemia
Brain Research · 1982 · 2,850 citations
Glutamate and the pathophysiology of hypoxic–ischemic brain damage
Annals of Neurology · 1986 · 2,432 citations
Glutamate: A Neurotransmitter in Mammalian Brain
Journal of Neurochemistry · 1984 · 1,984 citations
Hypoxanthine as an Indicator of Hypoxia: Its Role in Health and Disease through Free Radical Production
Pediatric Research · 1988 · 416 citations
Glutamate becomes neurotoxic via the N-methyl-d-aspartate receptor when intracellular energy levels are reduced
Brain Research · 1988 · 976 citations
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Experimental Biology of Cerebral Hypoxia-Ischemia: Relation to Perinatal Brain Damage
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