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A genome-wide association study implicates diacylglycerol kinase eta (DGKH) and several other genes in the etiology of bipolar disorder

Molecular Psychiatry · 2007 · Vol. 13(2) · pp. 197–207

Amber E. Baum✉(National Institute of Mental Health)Nirmala Akula(United States Department of Health and Human Services)Michael Cabanero(National Institutes of Health)Imer Cardona(National Institutes of Health)Winston Corona(United States Department of Health and Human Services)Brygida Klemens(Brookings Institution)Thomas G. Schulze(Central Institute of Mental Health)Sven Cichon(Life & Brain (Germany))Marcella Rietschel(Central Institute of Mental Health)Markus M. Nöthen(Life & Brain (Germany))Alexander Georgi(Heidelberg University)J Schumacher(University of Bonn)Markus Schwarz(University Hospital Heidelberg)Rami Abou Jamra(University of Bonn)Susanne Höfels(University of Bonn)Peter Propping(University of Bonn)Jaya M. Satagopan(Memorial Sloan Kettering Cancer Center)Sevilla D. Detera‐Wadleigh(National Institutes of Health)John Hardy(National Institutes of Health)Francis J. McMahon(National Institute of Mental Health)

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Bipolar Disorder and Treatment Genetic Associations and Epidemiology Genetic Syndromes and ImprintingSingle-nucleotide polymorphismGenome-wide association studyGenetic associationBipolar disorderGeneticsBiologyGenotypingSNP genotypingGeneSNP

MeSH terms

Bipolar DisorderEuropeFemaleHumansMaleModels, BiologicalRandom AllocationLinkage DisequilibriumCase-Control StudiesGenomeDiacylglycerol KinaseGenetic Predisposition to DiseasePolymorphism, Single Nucleotide

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