articleTop 10% cited
White Cell Telomere Length and Risk of Premature Myocardial Infarction
Arteriosclerosis Thrombosis and Vascular Biology · 2003 · Vol. 23(5) · pp. 842–846
Scott Brouilette✉(University of Leicester)Ravi Singh(University of Leicester)John R. Thompson(University of Leicester)Alison H. Goodall(University of Leicester)Nilesh J. Samani(University of Leicester)
Abstract
The findings support the concept that biological age may play a role in the etiology of coronary heart disease and have potentially important implications for our understanding of its genetic etiology, pathogenesis, and variable age of onset.
Telomeres, Telomerase, and SenescenceBirth, Development, and HealthGenetics, Aging, and Longevity in Model OrganismsTelomerePathogenesisMyocardial infarctionEtiologyInternal medicineMedicineQuartileBiological ageCardiologyPhysiology
MeSH terms
AdultAgingCoronary DiseaseEnglandFemaleHumansLeukocytesMaleMiddle AgedMyocardial InfarctionRetrospective StudiesRiskRisk FactorsTelomereAge of Onset
Funding
- Medical Research Council
Citations
590
FWCI
9.18
field-weighted impact
References
28
Percentile
99%
vs. same field & year
Citations per year
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References
A theory of marginotomy
Journal of Theoretical Biology · 1973 · 1,816 citations
Heterogeneity in telomere length of human chromosomes
Human Molecular Genetics · 1996 · 878 citations
Atherosclerosis — An Inflammatory Disease
New England Journal of Medicine · 1999 · 21,632 citations
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