articleTop 10% cited
Glial cytoplasmic inclusions in the CNS of patients with multiple system atrophy (striatonigral degeneration, olivopontocerebellar atrophy and Shy-Drager syndrome)
Journal of the Neurological Sciences · 1989 · Vol. 94(1-3) · pp. 79–100
M Papp✉(Semmelweis University)J. Kahn(Institute of Ophthalmology)P. L. Lantos(Institute of Ophthalmology)
Neurological diseases and metabolismAlzheimer's disease research and treatmentsParkinson's Disease Mechanisms and TreatmentsOlivopontocerebellar atrophyPathologyAtrophyImmunocytochemistryBiologyEpendymaMyelinCentral nervous systemCytoplasmic inclusionDegenerative disease
MeSH terms
AdultAgedAged, 80 and overAutonomic Nervous System DiseasesInclusion BodiesCorpus StriatumFemaleHumansMaleMiddle AgedNeurogliaOlivopontocerebellar AtrophiesShy-Drager SyndromeSpinocerebellar DegenerationsSubstantia Nigra
Citations
990
FWCI
6.68
field-weighted impact
References
32
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97%
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Citations per year
Cited by
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α-Synuclein immunoreactivity in glial cytoplasmic inclusions in multiple system atrophy
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Movement Disorders · 2003 · 1,018 citations
Filamentous α-synuclein inclusions link multiple system atrophy with Parkinson's disease and dementia with Lewy bodies
Neuroscience Letters · 1998 · 1,027 citations
Multiple system atrophy: A review of 203 pathologically proven cases
Movement Disorders · 1997 · 794 citations
References
Greenfield's neuropathology
Journal of the Neurological Sciences · 1976 · 966 citations
Ubiquitin is a common factor in intermediate filament inclusion bodies of diverse type in man, including those of Parkinson's disease, Pick's disease, and Alzheimer's disease, as well as Rosenthal fibres in cerebellar astrocytomas, cytoplasmic bodies in muscle, and mallory bodies in alcoholic liver disease
The Journal of Pathology · 1988 · 542 citations
Greenfield's Neuropathology
Journal of Neurology Neurosurgery & Psychiatry · 1985 · 1,156 citations
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