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Recurrent DNMT3A mutations in patients with myelodysplastic syndromes

Leukemia · 2011 · Vol. 25(7) · pp. 1153–1158

Matthew J. Walter✉(Washington University in St. Louis)Li Ding(Washington University in St. Louis)Dong Shen(Washington University in St. Louis)Jieya Shao(Washington University in St. Louis)Marcus Grillot(Washington University in St. Louis)Michael D. McLellan(Washington University in St. Louis)Robert S. Fulton(Washington University in St. Louis)H Schmidt(Washington University in St. Louis)Joelle Kalicki-Veizer(Washington University in St. Louis)Michele O’Laughlin(Washington University in St. Louis)Cyriac Kandoth(Washington University in St. Louis)Jack Baty(Washington University in St. Louis)Peter Westervelt(Washington University in St. Louis)John F. DiPersio(Washington University in St. Louis)Elaine R. Mardis(Washington University in St. Louis)Richard K. Wilson(Washington University in St. Louis)T J Ley(Washington University in St. Louis)Timothy A. Graubert(Washington University in St. Louis)

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Acute Myeloid Leukemia Research Epigenetics and DNA Methylation Hemoglobinopathies and Related DisordersMyelodysplastic syndromesMethyltransferaseDNMT3BMutationBiologyCancer researchDNA methylationMyeloid leukemiaDNA methyltransferaseMethylation

MeSH terms

DNA Methyltransferase 3AAdultAgedAged, 80 and overCodonDNA (Cytosine-5-)-MethyltransferasesDNA, NeoplasmExonsFemaleHumansMaleMiddle AgedMutationMyelodysplastic SyndromesPrognosis

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Clinical and biological implications of driver mutations in myelodysplastic syndromes