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Large and Sustained Induction of Chemokines during Impaired Wound Healing in the Genetically Diabetic Mouse: Prolonged Persistence of Neutrophils and Macrophages during the Late Phase of Repair

Journal of Investigative Dermatology · 2000 · Vol. 115(2) · pp. 245–253

Christian Wetzler(Goethe University Frankfurt)Heiko Kämpfer(Goethe University Frankfurt)Birgit Stallmeyer(Goethe University Frankfurt)Josef Pfeilschifter(Goethe University Frankfurt)Stefan L. Frank✉(Goethe University Frankfurt)

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Wound Healing and Treatments Immune Response and Inflammation NF-κB Signaling PathwaysChemokineMacrophage inflammatory proteinWound healingMacrophageChemotaxisImmunologyInfiltration (HVAC)InflammationBiologyCell biology

MeSH terms

AnimalsDermatitisDiabetes MellitusMacrophagesMice, Inbred C57BLNeutrophilsSkinTime FactorsWound HealingWounds and InjuriesDown-RegulationKeratinocytesReceptors, InterleukinChemokinesChemokine CCL2

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576

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12.23

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99%

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Topical Vascular Endothelial Growth Factor Accelerates Diabetic Wound Healing through Increased Angiogenesis and by Mobilizing and Recruiting Bone Marrow-Derived Cells