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Delayed increase of Ca2+ influx elicited by glutamate: role in neuronal death.

Molecular Pharmacology · 1989 · Vol. 36(1) · pp. 106–112
Hari ManevMarco FavaronAlessandro GuidottiE. Costa
Neuroscience and Neuropharmacology ResearchMitochondrial Function and PathologyIon channel regulation and functionGlutamate receptorKainate receptorNMDA receptorNeurotoxicityChemistryPharmacologyBiologyReceptorBiochemistryToxicity

MeSH terms

AnimalsAspartic AcidBiological TransportCalciumCell SurvivalCells, CulturedGlutamatesNeuronsProtein Kinase CQuinoxalinesRats, Inbred StrainsN-MethylaspartateReceptors, GlutamateReceptors, NeurotransmitterGlutamic Acid
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Delayed increase of Ca2+ influx elicited by glutamate: role in neuronal death. · Scinovex